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Steven L. Bealer

Professor of Pharmacology and Toxicology

Education and Training

B.S. 1971, University of Wyoming

Ph.D. 1976, University of Wyoming

Research Interests

One series of ongoing studies is directed at understanding the effects of seizure disorders on heart function and the increased risk of sudden cardiac death, using an animal model of status epilepticus and temporal lobe epilepsy. A separate line of investigation is aimed at understanding the mechanisms through which these seizure disorders disrupt ovarian hormone secretion and energy balance in female rats.

Selected Publications

Lipschitz, D.L., Crowley, R.W. and Bealer, S.L. Differential sensitivity of intranuclear and systemic oxytocin release to central noradrenergic receptor stimulation during mid- and late gestation in rats. American Journal of Physiology (Endocrinology and Metabolism) 287: E523-E528, 2004.

Bealer, S.L. and Metcalf, C.S.. Increased dietary sodium enhances activation of neurons in the medullary cardiovascular pathway during acute sodium loading in the rat. Autonomic Neuroscience 117: 33-40, 2005.

Bealer, S.L. Increased dietary sodium inhibits baroreflex-induced bradycardia during acute sodium   loading.  American Journal of Physiology (Regulatory, Integrative and Comparative Physiology). 288: R1211-R1219, 2005.

Bealer, S.L., Lipschitz, D.L., Ramoz, G., Crowley, R.W. Oxytocin receptor binding in magnocellular nuclei during gestation in rats. American Journal of Physiology: Reg., Int., and Comp. Physiol. 291, R53-R58, 2006.

Bealer, S.L., Metcalf, C.S., and Heyborne, R. Increased dietary sodium alters Fos expression in the lamina terminalis during intravenous angiotensin II infusion. Experimental Neurology 204, 299-306, 2007.

Pharmacology & Toxicology   College of Pharmacy   30 S. 2000 E., Rm 201, SLC  UT 84112  phone: 801-581-6287  fax: 801-585-5111 phtx@pharm.utah.edu